![]() People also reported increased difficulties falling asleep, poorer sleep quality, more restless sleep and more "lighter" sleep.Ī more recent study found patients with COVID-19 reported more trouble sleeping compared to patients without COVID. One study that looked at rhinovirus infections - or the "common cold" - in healthy adults, found individuals who are symptomatic had a reduced sleep duration, less consolidated sleep, and poorer cognitive performance than asymptomatic individuals.Īnother study that looked at people with respiratory infections showed that while symptomatic, people spent more time in bed and had increased sleep time, yet had more awakenings during sleep. As such, sleep increases the immune response which may increase our chance of survival from the infection. This means sleep, in particular poor sleep, can impact immune function, and immune function can impact sleep.ĭuring sleep, especially during the non-rapid eye movement stage slow-wave sleep (a deep stage of sleep), there is an increase in the production of some cytokines. ![]() It gets a little more complicated, though, because like many things, sleep and immunity are bidirectional. In this way, when there are more of these cytokines in our bodies this tends to make us sleepier. Some of these cytokines are also involved in promoting sleep and are known as " sleep regulatory substances". As part of this response, our cells produce proteins such as cytokines in order to help fight the infection.\ When our body is infected with a virus this causes an immune, or inflammatory response. So why is our sleep impacted by COVID infections, and why do the impacts differ so much between individuals? Sleep and immunity
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